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Azilsartan Suppressed LPS-Induced Inflammation in U937 Macrophages through  Suppressing Oxidative Stress and Inhibiting the TLR2/MyD88 Signal Pathway |  ACS Omega
Azilsartan Suppressed LPS-Induced Inflammation in U937 Macrophages through Suppressing Oxidative Stress and Inhibiting the TLR2/MyD88 Signal Pathway | ACS Omega

Lipopolysaccharides induced inflammatory responses and electrophysiological  dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes  | Scientific Reports
Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes | Scientific Reports

Neuroinflammation induced by lipopolysaccharide causes cognitive impairment  in mice | Scientific Reports
Neuroinflammation induced by lipopolysaccharide causes cognitive impairment in mice | Scientific Reports

LPS‑induced proinflammatory cytokine expression in human airway epithelial  cells and macrophages via NF‑κB, STAT3 or AP‑1 activation
LPS‑induced proinflammatory cytokine expression in human airway epithelial cells and macrophages via NF‑κB, STAT3 or AP‑1 activation

Surfactant Protein A Enhances the Degradation of LPS-Induced TLR4 in  Primary Alveolar Macrophages Involving Rab7, β-arrestin2, and mTORC1 |  Infection and Immunity
Surfactant Protein A Enhances the Degradation of LPS-Induced TLR4 in Primary Alveolar Macrophages Involving Rab7, β-arrestin2, and mTORC1 | Infection and Immunity

Surfactant Protein A Enhances the Degradation of LPS-Induced TLR4 in  Primary Alveolar Macrophages Involving Rab7, β-arrestin2, and mTORC1 |  Infection and Immunity
Surfactant Protein A Enhances the Degradation of LPS-Induced TLR4 in Primary Alveolar Macrophages Involving Rab7, β-arrestin2, and mTORC1 | Infection and Immunity

Lipopolysaccharide-Induced Microglial Activation and Neuroprotection  against Experimental Brain Injury Is Independent of Hematogenous TLR4 |  Journal of Neuroscience
Lipopolysaccharide-Induced Microglial Activation and Neuroprotection against Experimental Brain Injury Is Independent of Hematogenous TLR4 | Journal of Neuroscience

Effect of LPS-treatment on pro-inflammatory cytokine/chemokine levels.... |  Download Scientific Diagram
Effect of LPS-treatment on pro-inflammatory cytokine/chemokine levels.... | Download Scientific Diagram

A Novel Anti-inflammatory Phenotype Transformed by Repetitive Low-dose  Lipopolysaccharide in Primary Peritoneal Tissue-resident Macrophages |  Anticancer Research
A Novel Anti-inflammatory Phenotype Transformed by Repetitive Low-dose Lipopolysaccharide in Primary Peritoneal Tissue-resident Macrophages | Anticancer Research

Lipopolysaccharide (LPS)-Induced Biliary Epithelial Cell NRas Activation  Requires Epidermal Growth Factor Receptor (EGFR) | PLOS ONE
Lipopolysaccharide (LPS)-Induced Biliary Epithelial Cell NRas Activation Requires Epidermal Growth Factor Receptor (EGFR) | PLOS ONE

Life | Free Full-Text | Natural Sulfurs Inhibit LPS-Induced Inflammatory  Responses through NF-κB Signaling in CCD-986Sk Skin Fibroblasts
Life | Free Full-Text | Natural Sulfurs Inhibit LPS-Induced Inflammatory Responses through NF-κB Signaling in CCD-986Sk Skin Fibroblasts

IJMS | Free Full-Text | Alleviation of LPS-Induced Inflammation and Septic  Shock by Lactiplantibacillus plantarum K8 Lysates
IJMS | Free Full-Text | Alleviation of LPS-Induced Inflammation and Septic Shock by Lactiplantibacillus plantarum K8 Lysates

Ginsenoside Rg1 attenuates LPS-induced cognitive impairments and  neuroinflammation by inhibiting NOX2 and Ca2+–CN–NFAT1 signaling in mice -  ScienceDirect
Ginsenoside Rg1 attenuates LPS-induced cognitive impairments and neuroinflammation by inhibiting NOX2 and Ca2+–CN–NFAT1 signaling in mice - ScienceDirect

A unique hybrid characteristic having both pro- and anti-inflammatory  phenotype transformed by repetitive low-dose lipopolysaccharide in C8-B4  microglia | Scientific Reports
A unique hybrid characteristic having both pro- and anti-inflammatory phenotype transformed by repetitive low-dose lipopolysaccharide in C8-B4 microglia | Scientific Reports

Lipopolysaccharides induced inflammatory responses and electrophysiological  dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes  | Scientific Reports
Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes | Scientific Reports

The LPS-Induced Transcriptional Upregulation of the Chicken Lysozyme Locus  Involves CTCF Eviction and Noncoding RNA Transcription: Molecular Cell
The LPS-Induced Transcriptional Upregulation of the Chicken Lysozyme Locus Involves CTCF Eviction and Noncoding RNA Transcription: Molecular Cell

Effect of LPS treatment of RB family protein expression. Western... |  Download Scientific Diagram
Effect of LPS treatment of RB family protein expression. Western... | Download Scientific Diagram

Lipopolysaccharides induced inflammatory responses and electrophysiological  dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes  | Scientific Reports
Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes | Scientific Reports

SRV2 promotes mitochondrial fission and Mst1-Drp1 signaling in LPS-induced  septic cardiomyopathy | Aging
SRV2 promotes mitochondrial fission and Mst1-Drp1 signaling in LPS-induced septic cardiomyopathy | Aging

Frontiers | A Novel Lipopolysaccharide Recognition Mechanism Mediated by  Internalization in Teleost Macrophages
Frontiers | A Novel Lipopolysaccharide Recognition Mechanism Mediated by Internalization in Teleost Macrophages

Dexmedetomidine inhibits LPS-induced proinflammatory responses via  suppressing HIF1α-dependent glycolysis in macrophages | Aging
Dexmedetomidine inhibits LPS-induced proinflammatory responses via suppressing HIF1α-dependent glycolysis in macrophages | Aging

Lipopolysaccharide induces inflammation and facilitates lung metastasis in  a breast cancer model via the prostaglandin E2-EP2 pathway
Lipopolysaccharide induces inflammation and facilitates lung metastasis in a breast cancer model via the prostaglandin E2-EP2 pathway

Inhibition of Lipopolysaccharide-Induced Inflammatory Responses by an  Apolipoprotein AI Mimetic Peptide | Circulation Research
Inhibition of Lipopolysaccharide-Induced Inflammatory Responses by an Apolipoprotein AI Mimetic Peptide | Circulation Research