![Azilsartan Suppressed LPS-Induced Inflammation in U937 Macrophages through Suppressing Oxidative Stress and Inhibiting the TLR2/MyD88 Signal Pathway | ACS Omega Azilsartan Suppressed LPS-Induced Inflammation in U937 Macrophages through Suppressing Oxidative Stress and Inhibiting the TLR2/MyD88 Signal Pathway | ACS Omega](https://pubs.acs.org/cms/10.1021/acsomega.0c03655/asset/images/medium/ao0c03655_0009.gif)
Azilsartan Suppressed LPS-Induced Inflammation in U937 Macrophages through Suppressing Oxidative Stress and Inhibiting the TLR2/MyD88 Signal Pathway | ACS Omega
![Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes | Scientific Reports Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes | Scientific Reports](https://media.springernature.com/lw685/springer-static/image/art%3A10.1038%2Fs41598-017-03147-4/MediaObjects/41598_2017_3147_Fig3_HTML.jpg)
Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes | Scientific Reports
![Neuroinflammation induced by lipopolysaccharide causes cognitive impairment in mice | Scientific Reports Neuroinflammation induced by lipopolysaccharide causes cognitive impairment in mice | Scientific Reports](https://media.springernature.com/lw685/springer-static/image/art%3A10.1038%2Fs41598-019-42286-8/MediaObjects/41598_2019_42286_Fig7_HTML.png)
Neuroinflammation induced by lipopolysaccharide causes cognitive impairment in mice | Scientific Reports
![LPS‑induced proinflammatory cytokine expression in human airway epithelial cells and macrophages via NF‑κB, STAT3 or AP‑1 activation LPS‑induced proinflammatory cytokine expression in human airway epithelial cells and macrophages via NF‑κB, STAT3 or AP‑1 activation](https://www.spandidos-publications.com/article_images/mmr/17/4/MMR-17-04-5484-g01.jpg)
LPS‑induced proinflammatory cytokine expression in human airway epithelial cells and macrophages via NF‑κB, STAT3 or AP‑1 activation
![Surfactant Protein A Enhances the Degradation of LPS-Induced TLR4 in Primary Alveolar Macrophages Involving Rab7, β-arrestin2, and mTORC1 | Infection and Immunity Surfactant Protein A Enhances the Degradation of LPS-Induced TLR4 in Primary Alveolar Macrophages Involving Rab7, β-arrestin2, and mTORC1 | Infection and Immunity](https://journals.asm.org/cms/10.1128/iai.00250-21/asset/d8b6cb62-a966-4ff1-9c74-c9f3078fbde2/assets/images/large/iai.00250-21-f001b.jpg)
Surfactant Protein A Enhances the Degradation of LPS-Induced TLR4 in Primary Alveolar Macrophages Involving Rab7, β-arrestin2, and mTORC1 | Infection and Immunity
![Surfactant Protein A Enhances the Degradation of LPS-Induced TLR4 in Primary Alveolar Macrophages Involving Rab7, β-arrestin2, and mTORC1 | Infection and Immunity Surfactant Protein A Enhances the Degradation of LPS-Induced TLR4 in Primary Alveolar Macrophages Involving Rab7, β-arrestin2, and mTORC1 | Infection and Immunity](https://journals.asm.org/cms/10.1128/iai.00250-21/asset/67b2ca68-4dcc-454f-a30e-811cebf7bcc1/assets/images/large/iai.00250-21-f001a.jpg)
Surfactant Protein A Enhances the Degradation of LPS-Induced TLR4 in Primary Alveolar Macrophages Involving Rab7, β-arrestin2, and mTORC1 | Infection and Immunity
![Lipopolysaccharide-Induced Microglial Activation and Neuroprotection against Experimental Brain Injury Is Independent of Hematogenous TLR4 | Journal of Neuroscience Lipopolysaccharide-Induced Microglial Activation and Neuroprotection against Experimental Brain Injury Is Independent of Hematogenous TLR4 | Journal of Neuroscience](https://www.jneurosci.org/content/jneuro/32/34/11706/F2.large.jpg)
Lipopolysaccharide-Induced Microglial Activation and Neuroprotection against Experimental Brain Injury Is Independent of Hematogenous TLR4 | Journal of Neuroscience
Effect of LPS-treatment on pro-inflammatory cytokine/chemokine levels.... | Download Scientific Diagram
![A Novel Anti-inflammatory Phenotype Transformed by Repetitive Low-dose Lipopolysaccharide in Primary Peritoneal Tissue-resident Macrophages | Anticancer Research A Novel Anti-inflammatory Phenotype Transformed by Repetitive Low-dose Lipopolysaccharide in Primary Peritoneal Tissue-resident Macrophages | Anticancer Research](https://ar.iiarjournals.org/content/anticanres/40/8/4457/F4.large.jpg)
A Novel Anti-inflammatory Phenotype Transformed by Repetitive Low-dose Lipopolysaccharide in Primary Peritoneal Tissue-resident Macrophages | Anticancer Research
Lipopolysaccharide (LPS)-Induced Biliary Epithelial Cell NRas Activation Requires Epidermal Growth Factor Receptor (EGFR) | PLOS ONE
![Life | Free Full-Text | Natural Sulfurs Inhibit LPS-Induced Inflammatory Responses through NF-κB Signaling in CCD-986Sk Skin Fibroblasts Life | Free Full-Text | Natural Sulfurs Inhibit LPS-Induced Inflammatory Responses through NF-κB Signaling in CCD-986Sk Skin Fibroblasts](https://www.mdpi.com/life/life-11-00427/article_deploy/html/images/life-11-00427-g001.png)
Life | Free Full-Text | Natural Sulfurs Inhibit LPS-Induced Inflammatory Responses through NF-κB Signaling in CCD-986Sk Skin Fibroblasts
![IJMS | Free Full-Text | Alleviation of LPS-Induced Inflammation and Septic Shock by Lactiplantibacillus plantarum K8 Lysates IJMS | Free Full-Text | Alleviation of LPS-Induced Inflammation and Septic Shock by Lactiplantibacillus plantarum K8 Lysates](https://www.mdpi.com/ijms/ijms-22-05921/article_deploy/html/images/ijms-22-05921-g005.png)
IJMS | Free Full-Text | Alleviation of LPS-Induced Inflammation and Septic Shock by Lactiplantibacillus plantarum K8 Lysates
![Ginsenoside Rg1 attenuates LPS-induced cognitive impairments and neuroinflammation by inhibiting NOX2 and Ca2+–CN–NFAT1 signaling in mice - ScienceDirect Ginsenoside Rg1 attenuates LPS-induced cognitive impairments and neuroinflammation by inhibiting NOX2 and Ca2+–CN–NFAT1 signaling in mice - ScienceDirect](https://ars.els-cdn.com/content/image/1-s2.0-S1756464621004400-gr4.jpg)
Ginsenoside Rg1 attenuates LPS-induced cognitive impairments and neuroinflammation by inhibiting NOX2 and Ca2+–CN–NFAT1 signaling in mice - ScienceDirect
![A unique hybrid characteristic having both pro- and anti-inflammatory phenotype transformed by repetitive low-dose lipopolysaccharide in C8-B4 microglia | Scientific Reports A unique hybrid characteristic having both pro- and anti-inflammatory phenotype transformed by repetitive low-dose lipopolysaccharide in C8-B4 microglia | Scientific Reports](https://media.springernature.com/m685/springer-static/image/art%3A10.1038%2Fs41598-020-65998-8/MediaObjects/41598_2020_65998_Fig1_HTML.png)
A unique hybrid characteristic having both pro- and anti-inflammatory phenotype transformed by repetitive low-dose lipopolysaccharide in C8-B4 microglia | Scientific Reports
![Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes | Scientific Reports Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes | Scientific Reports](https://media.springernature.com/lw685/springer-static/image/art%3A10.1038%2Fs41598-017-03147-4/MediaObjects/41598_2017_3147_Fig2_HTML.jpg)
Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes | Scientific Reports
![The LPS-Induced Transcriptional Upregulation of the Chicken Lysozyme Locus Involves CTCF Eviction and Noncoding RNA Transcription: Molecular Cell The LPS-Induced Transcriptional Upregulation of the Chicken Lysozyme Locus Involves CTCF Eviction and Noncoding RNA Transcription: Molecular Cell](https://www.cell.com/cms/attachment/a336fd00-ed08-4d62-b1de-0ddc56259ca1/gr1.jpg)
The LPS-Induced Transcriptional Upregulation of the Chicken Lysozyme Locus Involves CTCF Eviction and Noncoding RNA Transcription: Molecular Cell
![Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes | Scientific Reports Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes | Scientific Reports](https://media.springernature.com/full/springer-static/image/art%3A10.1038%2Fs41598-017-03147-4/MediaObjects/41598_2017_3147_Fig1_HTML.jpg)
Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes | Scientific Reports
SRV2 promotes mitochondrial fission and Mst1-Drp1 signaling in LPS-induced septic cardiomyopathy | Aging
![Frontiers | A Novel Lipopolysaccharide Recognition Mechanism Mediated by Internalization in Teleost Macrophages Frontiers | A Novel Lipopolysaccharide Recognition Mechanism Mediated by Internalization in Teleost Macrophages](https://www.frontiersin.org/files/Articles/421138/fimmu-09-02758-HTML/image_m/fimmu-09-02758-g001.jpg)
Frontiers | A Novel Lipopolysaccharide Recognition Mechanism Mediated by Internalization in Teleost Macrophages
Dexmedetomidine inhibits LPS-induced proinflammatory responses via suppressing HIF1α-dependent glycolysis in macrophages | Aging
![Lipopolysaccharide induces inflammation and facilitates lung metastasis in a breast cancer model via the prostaglandin E2-EP2 pathway Lipopolysaccharide induces inflammation and facilitates lung metastasis in a breast cancer model via the prostaglandin E2-EP2 pathway](https://www.spandidos-publications.com/article_images/mmr/11/6/MMR-11-06-4454-g03.jpg)
Lipopolysaccharide induces inflammation and facilitates lung metastasis in a breast cancer model via the prostaglandin E2-EP2 pathway
![Inhibition of Lipopolysaccharide-Induced Inflammatory Responses by an Apolipoprotein AI Mimetic Peptide | Circulation Research Inhibition of Lipopolysaccharide-Induced Inflammatory Responses by an Apolipoprotein AI Mimetic Peptide | Circulation Research](https://www.ahajournals.org/cms/asset/899f9aa9-5b35-47e1-93f8-62415b5d5c84/7ff5.jpeg)